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| (nal-ih-DIK-sik acid) |
| NegGram |
| Class: Urinary anti-infective |
Action Interferes with DNA formation of certain bacteria.
Indications Treatment of UTIs caused by susceptible gram-negative bacteria, including most Proteus strains, Klebsiella and Enterobacter species and E. coli.
Contraindications History of seizures.
ADULTS: PO Initial therapy: 1 g qid for 1 or 2 wk. Prolonged Therapy: 1 g bid after initial therapy. Children 3 mo to 2 YR: PO Initial therapy: 55 mg/kg/day divided into 4 equal doses. Prolonged Therapy: 33 mg/kg/day in 4 divided doses after initial therapy.
Oral anticoagulants: May enhance anticoagulant effect.
Lab Test Interferences False-positive urinary glucose results with Benedict’s or Fehling’s solutions or Clinitest reagent tablets; use Clinistix or Tes-tape. Urinary 17-keto and ketogenic steroids may be falsely elevated; Porter-Silber method should be used.
CNS: Drowsiness; weakness; headache; dizziness; vertigo; seizures; intracranial hypertension; increased intracranial pressure; sixth cranial nerve palsy in children and infants. DERM: Rash; pruritus; urticaria; angioedema; photosensitivity. EENT: Visual disturbances. GI: Abdominal pain; nausea; vomiting; diarrhea. HEMA: Thrombocytopenia; leukopenia, eosinophilia or hemolytic anemia (associated with G-6-PD deficiency or acute nalidixi reaction). HEPA: Cholestatic jaundice; cholestasis. META: Metabolic acidosis.
Pregnancy: Pregnancy category undetermined. Do not use during first trimester. Lactation: Excreted in breast milk. Children: Use drug with caution in prepubertal children; may affect cartilage and joints. CNS effects: Convulsions, increased intracranial pressure and toxic psychosis may occur with overdose or predisposing factors (eg, epilepsy, cerebral arteriosclerosis). Hematologic: Can produce clinically significant hemolysis in patients with G-6-PD deficiency. Renal failure: Patients with compromised renal function may fail to accumulate nalidixic acid, decreasing its effectiveness.
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